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Infant girls with evidence of exposure to the CoVid virus were more than twice as likely to have antibodies against their own insulin-producing pancreatic islet cells as those without traces of CoVid in their systems. German investigators led a multi-center collaborative cohort study of 885 female infants with a genetic predisposition to develop insulin-dependent type 1 diabetes.
The children were followed from 6 to 25 months of age and repeatedly tested for antibodies to CoVid and antibodies to their islet cells. The data revealed that the incidence rate of islet autoantibodies was 7.8 per 100-person years in those kids with coexistent CoVid antibodies but only 3.5 per 100-person years in those without immunologic evidence of CoVid exposure.
Other studies have suggested an association between CoVid infection and autoimmunity. Another large German study revealed that CoVid patients were 42% more likely to develop autoimmune diseases including thyroiditis, rheumatoid arthritis, and salivary gland inflammation.
These studies are just some of the increasing evidence that Covid’s propensity to hyper-stimulate the human immune system may be a chief mechanism by which this family of coronaviruses drives continuing disability long after the acute infection has resolved. Inn other words: Long CoVid.
https://jamanetwork.com/journals/jama/fullarticle/2809621
https://pubmed.ncbi.nlm.nih.gov/37335408/
#CoVid #longcovid #diabetes #autoimmunity #thyroiditis #arthritis